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DESIRE CLIMAX PDF

Wednesday, June 5, 2019


From ShoujoMagic: Omori Mio is 17 years old. To help out her desperately poor family, she takes on a part-time job. Then, one night, a boy. This Pin was discovered by Patricija Hasler. Discover (and save) your own Pins on Pinterest. Desire Climax is a shōjo manga series by Ayane Ukyō. It was serialized in Shueisha's Margaret . Create a book · Download as PDF · Printable version.


Desire Climax Pdf

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Ahhhhhhhh my favorite manga. ^ ^ Nothing beats well almost nothing beats this series. Anyways, if your looking for the translated chapters. Desire Climax (Tomo 1). By Manga Shôjo · Updated about 6 years ago. Already tagged. 5. Already tagged · Already tagged. 2. Already tagged. 4. Download pdf book by Ayane Ukyou - Free eBooks.

From Wikipedia, the free encyclopedia. Desire Climax Cover art from volume 1 of the manga Desire Climax.

Planet Manga. Retrieved 3 April Egmont Manga. Retrieved 10 April Manga News. Retrieved 25 August Retrieved from " https: Hidden categories: Vascular supply Blood supply to the body of the clitoris is supplied by paired pudendal arteries that course lateral to the urethra along the pelvic aspect of the anterior vaginal wall [ 1 ].

These arteries give off the cavernous artery and the artery to the bulb. The body of the clitoris is composed of cavernous tissue surrounded by tough tunica albuginea.

Pls tell me the easiest way to download Desire Climax Manga chapters?..?

However, the clitoris seems to lack the subalbugineal plexus characteristic of the penis [ 4 ]. This finding suggests that, unlike the penis, the clitoris does not have a well developed outflow occlusion mechanism and becomes erect mainly through increased blood flow. Innervation and histochemistry The glans of the clitoris forms a cap that sits on the ends of the corporal bodies and is richly innervated by sensory nerve endings [ 5 ].

The deep dorsal nerves perforate the glans on the dorsal aspect of its junction with the corporal bodies.

Recent studies have confirmed that the dorsal nerve of the clitoris is relatively large more than 2 mm in diameter [ 1 ]. The cavernous nerve runs along with the cavernous artery as it enters the bodies of the clitoris. Nerves containing neuronal nitric oxide synthase have been detected in both the body and the glans of the human clitoris [ 6 ], suggesting that nitric oxide is involved in the control of clitoral smooth muscle tone.

Recent anatomic studies have shown that these erectile bodies are intimately related and lateral to the urethra rather than lateral to the vestibule of the vagina [ 1 ].

Desire Climax

They are not, as often depicted in anatomic texts, located within the labia minora. Superiorly, the bulbs continue as the pars intermedia and terminate as the glans of the clitoris [ 9 ]. Thus, the bulbs represent the cleft female homologue to the bulb of the urethra in the male. The entire complex of spongy erectile tissue consisting of bulbs, pars intermedia, and clitoral glans correspond to the corpus spongiosum of the male [ 9 ].

Structure The vagina is a muscular sheath that connects the uterus and the external genitalia. It has both sexual and reproductive functions and this dual function is reflected in various aspects of its structure, especially its vascular supply, its sensory and motor innervation and its marked distensibility.

The wall of the vagina consists of an inner lining of stratified squamous epithelium, an intermediate layer of smooth muscle and an outer adventitial layer. The lining of the vagina is folded into numerous rugae which are related to its extreme distensibility.

The vaginal entrance is closed by the bulbocavernosus muscle, a striated muscle. Vascular supply Corrosion studies of the rat vagina have shown that the vaginal epithelium is intimately related to a dense capillary network in the subepithelial layer [ 10 ].

This vascular network may be related to the production of transudate during sexual arousal. Somewhat deeper, the loose submucosal layer contains a complex of numerous large veins and smooth muscle fibers that resemble cavernous tissue. The smooth muscle layer consists of an inner circular layer and a strong external longitudinal layer. The adventitial layer consists of connective tissue and a large plexus of blood vessels. Innervation and histochemistry The vagina has a complex innervation that is not well understood.

The pelvic nerves appear to subserve sensation from the vagina while the pudendal nerve subserves sensation from the labia and clitoris [ 11 ].

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Afferent nerves in the vagina appear to contain substance P in both animal [ 11 , 12 ] and human [ 13 ] tissue samples, although SP-containing nerves appear to be sparse in the human vagina [ 13 ]. Nerves containing nitric oxide synthase NOS have been demonstrated in animal [ 14 ] and human vaginas [ 15 ]. Neurovascular interactions during sexual arousal lead to several hemodynamic phases which affect simultaneously the clitoris, vestibular bulbs, and labia minora as well as the vagina.

In the resting phase, the vagina is a sheath containing a potential space with a minimal blood flow and very low oxygen tension in the wall [ 17 , 18 ]. The earliest detectable sign of sexual arousal based on studies with experimental models is a significant increase in vaginal wall and clitoral blood flow [ 19 ]. There is a significant increase in clitoral cavernosal pressure as well [ 19 ]. With the onset of increased vaginal blood flow, production of vaginal transudate ensues [ 10 , 20 ].

A significant rise in tissue oxygen tension follows about 20 seconds later which indicates increased inflow of arterial blood. In humans, vaginal and labial oxygen tension increases from 4 to 8 times baseline during sexual stimulation [ 17 , 18 ] and vaginal wall blood flow increases approximately threefold [ 21 - 23 ]. In humans, clitoral blood flow has been estimated to increase from 4 to 11 times baseline during sexual stimulation [ 24 ].

The increased blood flow reaches a plateau phase during which vaginal fluid transudate production continues. The final or resolution phase is characterized by the slow return of blood flow to baseline values. In women, up to minutes is required for vaginal oxygen tension to return to baseline [ 17 ]. It is believed that the increase of clitoral intracavernosal and vaginal wall blood flow results in part from a decrease in vascular resistance and relaxation of clitoral cavernosal and vaginal wall tissues.

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The precise identity of the neurotransmitters that control vaginal blood flow is currently unknown. Atropine does not affect the pelvic nerve-stimulated rise in vaginal blood flow in animals [ 19 , 25 ] or the rise in vaginal blood flow seen in human females [ 26 ]. This evidence suggests that muscarinic neurotransmission is probably not involved.

The ability of sildenafil to relax clitoral tissue and enhance nerve-stimulated vaginal blood flow has been reported [ 27 , 28 ]. This knowledge, of course, suggests the involvement of the NO-cyclic guanosine monophosphate cGMP pathway [ 27 , 29 ].

At the same time, it has been reported that administration of VIP, either intravenously or by injection in the vaginal wall, increases vaginal blood flow and induces vaginal fluid production [ 10 , 30 ]. Intravaginal prostaglandin E1 can also increase vaginal blood flow [ 32 ]. While some authors have stated that VIP is the primary neurotransmitter in the vaginal circulation [ 21 , 22 ], much more research in this area is required.

A study with a rabbit model has shown that systemic administration of apomorphine had no effect on basal clitoral intracavernosal and vaginal wall blood flow while significantly increasing blood inflow in these organs during nerve-stimulated vaginal and clitoral engorgement [ 32 ].

The precise mechanism by which apomorphine enhances nerve-stimulated but not basal clitoral intracavernosal and vaginal blood flow remains unknown.

Apomorphine at concentrations of 0. This may suggest the involvement of dopaminergic receptors in regulating the hemodynamic mechanism of clitoral and vaginal engorgement. Vaginal blood flow appears to undergo phasic shifts in conjunction with rapid eye movement REM sleep [ 33 ]. Central mechanisms Little is known about the central control of female sexual arousal.

Sexual stimulation activates specific areas of the central nervous system such as the medial preoptic region, the anterior hypothalamic region, and the related limbic hippocampal structures.

This stimulates transmission of signals via the parasympathetic and sympathetic pathways. The medial amygdala appears to be an important center that utilizes vasopressin as a central neurotransmitter [ 34 ].

Oxytocin is also clearly involved; oxytocin serum levels measured before and after sexual stimulation in 12 healthy women were significantly elevated [ 35 ]. Although intravenous apomorphine, a centrally acting dopaminergic agent, has been shown to cause increased peak clitoral and vaginal wall blood flow [ 32 ], the role of dopamine in female sexual behavior is not established.

Spinal mechanisms The mechanism of genital arousal and orgasm during sexual stimulation involve spinal cord reflexes that are mediated by genital afferents originating from the pudendal nerve. Interneurons mediating these reflexes are known to be in a column in the central portion of the spinal gray matter. The efferent arm of the spinal reflexes involves sympathetic, parasympathetic, and somatic activity [ 36 ]. Studies with a rabbit model have shown that electrical stimulation of the vaginal branch of the pelvic nerve increases clitoral intracavernosal pressure and blood flow, vaginal wall pressure and blood flow, and vaginal length [ 19 ].

Afferent signaling during sexual stimulation enters the spinal cord in the sacral segments, which is then transmitted to supraspinal sites via the spinothalamic and spinoreticular systems [ 36 ]. The spinothalamic pathway contains myelinated fibers that end in the posterolateral nucleus of the thalamus, from which the signals are relayed to the medial thalamus.

The precise reflex mechanisms of clitoral erection and vaginal engorgement are yet to be studied.

Pls tell me the easiest way to download Desire Climax Manga chapters?..?

Peripheral mechanisms The mechanism of vaginal engorgement during sexual arousal involves vasodilation and significant changes in vaginal tone. The vaginal tissue responds to sexual arousal by relaxing and lengthening [ 19 ]. Pelvic nerve-stimulated increased vaginal tone was abolished by atropine while vercuronium bromide, a striated muscle relaxant, prevented the subsequent fall in vaginal tension [ 37 ].

These results suggest that vaginal contraction may be under cholinergic control while vaginal lengthening or enlargement may relate to striated muscle contraction. How striated muscle contraction produces a decrease in vaginal pressure is unclear. It has been shown in humans that the distal part of the vagina contains more nerve fibers compared to the proximal part; likewise, the anterior vaginal wall is more densely innervated than the posterior wall [ 38 ].

Embryological origins of the proximal and distal vagina are also suggested to be different. The distal two-thirds of the vagina is suggested to be a derivative of the urogenital sinus whereas the proximal part is thought to originate from the uterovaginal primordium [ 39 ]. In the rabbit, stimulation of the vaginal branch of the pelvic nerve results in lengthening and dilation of the vagina that further leads to a lowering of vaginal luminal pressure and an increase in intravaginal wall pressure [ 19 ].

In rats, stimulation of the pelvic nerve causes a biphasic response in vaginal wall tension [ 37 ]. There is a rapid, short-lived increase in vaginal wall tension followed by a fall in tension to below the baseline value which is indicative of vaginal wall relaxation [ 19 , 37 ].

Organ bath studies with the rabbit vaginal tissue have shown that at baseline tension, electrical field stimulation EFS causes a biphasic contraction and relaxation response [ 40 ].

The alpha-2 adrenoceptor blocker yohimbine and the beta blocker propranolol were found to have little effect on vaginal tissue contraction in response to EFS and norepinephrine.

Prazosin seemed to be significantly effective in inhibiting these contractile responses, suggesting that vaginal tissue contraction may be primarily mediated by the alpha-1 adrenoceptor [ 40 ].

Relaxation of the vaginal tissue appears to involve a non-adrenergic non-cholinergic NANC mechanism. The nature of the vaginal NANC neurotransmission remains controversial. The rabbit vaginal tissue relaxes after exposure to acetylcholine Ach , vasoactive intestinal polypeptide VIP , papaverine, and the nitric oxide NO donor nitroprusside in a concentration dependent manner [ 40 ].

VIP was found to be more effective in relaxing the vaginal tissue than the clitoral tissue [ 40 ]. The NO precursor L-arginine and the nitric oxide synthase inhibitor nitro-L-arginine L-NNA appeared to have little effect on vaginal tissue relaxation.

Another study, however, found that NNA significantly inhibits the electrically-stimulated relaxation of rabbit vaginal tissue [ 41 ]. A VIP receptor antagonist has been shown to significantly increase the relaxation response to VIP while having little effect on EFS-induced relaxation of the vaginal tissue [ 40 ].

Desire Climax wallpaper and high quality picture gallery on Minitokyo. It was serialized in Shueisha's Margaret manga magazine from to , and collected in seven. Other Albums. Free and No Registration required for Desire Climax 1.

Discover ideas about Desire Climax. Heat 1.The good divine brings Arabella around to see that she should accept the novelistic requirement of universal instrumentality, and she subjects herself to his arguments, acting as a willing participant in her conversion.

What I am calling an axiomatic of desire that informs the novel-reading public is a consequence of what Scott Gordon sees as one of the precepts of anti-romance. Paulson, Ronald. Poison to Others, Energy to Pao. Fuchs, Barbara.